Octreotide
Octreotide (Sandostatin) — Somatostatin Analog
Evidence & Status
- FDA Approved
- Strong Human Evidence
- Strong
At a Glance
A long-acting synthetic somatostatin analogue used in FDA-approved treatment of acromegaly and symptomatic hormone excess from carcinoid tumors and VIPomas. It also has extensive research and off-label use across neuroendocrine and gastrointestinal medicine.
Plain English
Octreotide mimics somatostatin, a natural hormone that acts like a brake on the release of several other hormones. Its longer duration makes that biology clinically useful. It can suppress excess growth hormone in acromegaly and reduce hormone-driven symptoms such as severe flushing and diarrhea from certain neuroendocrine tumors.
Overview
Octreotide is a synthetic cyclic octapeptide analogue of somatostatin with a substantially longer duration of action than the endogenous hormone. It suppresses secretion of growth hormone and several gastrointestinal and pancreatic hormones. FDA-approved uses include acromegaly and symptomatic control of hormone secretion from metastatic carcinoid tumors and VIPomas. Long-acting depot formulations allow chronic therapy at multi-week dosing intervals.
Research Summary
Octreotide has established product-specific uses in the Sandostatin Injection and Sandostatin LAR Depot labels. The PROMID randomized trial and NCT00171873 apply specifically to octreotide LAR in metastatic well-differentiated midgut neuroendocrine tumors; they do not establish effects for immediate-release octreotide, other formulations, or all indications. Immediate-release and LAR claims, including dosing and administration, remain formulation-specific.
Research Areas
- Acromegaly
- Carcinoid tumors
- VIPoma
- Neuroendocrine tumors
Safety & Risks
Serious Risks
- Octreotide can reduce gallbladder contractility and bile secretion, leading to gallstones or biliary sludge; complications such as cholecystitis, cholangitis, and pancreatitis have been reported
- Octreotide can alter insulin, glucagon, and growth-hormone regulation and may cause either hyperglycemia or hypoglycemia
- Bradycardia, conduction abnormalities, and other ECG changes have been reported; patients with relevant cardiac disease or interacting medications may require monitoring
- Octreotide can suppress thyroid-stimulating hormone and may lead to hypothyroidism during prolonged therapy
- Reduced pancreatic enzyme and bile secretion can contribute to fat malabsorption and nutritional deficiencies during long-term treatment
Reported Side Effects
- Abdominal pain or cramping
- Nausea
- Diarrhea or loose stools
- Injection site reactions
- Headache
- Gallbladder sludge or gallstones
- Hyperglycemia or hypoglycemia
- Bradycardia
Legal Status by Region
- United States
- Sandostatin Injection and Sandostatin LAR Depot have separate U.S. labels, indications, and dosing; formulation-specific claims must remain product-specific.
- United Kingdom
- Product- and jurisdiction-specific.
- Australia
- Product- and jurisdiction-specific.
- Canada
- Product- and jurisdiction-specific.
Citations & Sources
- Placebo-controlled, double-blind, prospective, randomized study on the effect of octreotide LAR in the control of tumor growth in patients with metastatic neuroendocrine midgut tumors: a report from the PROMID Study Group
Rinke A et al. · Journal of Clinical Oncology · 2009
PMID 19704057 · DOI 10.1200/JCO.2009.22.8510
- SANDOSTATIN (octreotide acetate) Injection — Prescribing Information
2025
- SANDOSTATIN LAR DEPOT (octreotide acetate) — Prescribing Information
- Octreotide LAR in Patients With Metastatic Midgut Neuroendocrine Tumors
Registry NCT00171873